感染加速阿尔茨海默病进展！神经学大牛提出新假说

在这篇综述中，研究人员强调了感染及其产物在广告发病中的作用，并提出了A和感染性假说在广告发生发展中的协同假说。

淀粉样蛋白沉积本身可能不会导致神经变性，但会导致大脑对感染性物质神经毒性作用敏感性的增加。在这种机制下，感染衍生的PAMP诱导的神经变性可能会被隐藏起来，由于其往往发生在已经表现出明显的小胶质细胞增生和广告病理变化的大脑区域，这导致科学家们忽视了它们的作用。

对于广告患者和他们的家属而言，世界上最遥远的距离可能不仅仅是生与死，而是曾经最熟悉、最亲密的人在眼前，却想不起他（她)的名字。衷心希望广告的研究尽快出现突破性进展，找到相应的治疗靶点，给这些患者及其家庭带来曙光。

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